By Roger S. Kirby MD FRCS(Urol) FEBU
This can be a condensed model of the author's winning Atlas of Prostatic Disease. The emphasis is on a succinct description of prostatic affliction in a principally illustrative layout. The publication covers the motives, prognosis and staging of prostate melanoma, benign prostatic hyperplasia and prostatitis. The sections on remedy were more advantageous by means of the addition of algorithms to summarize the decision-making method.
Men's wellbeing and fitness is now an immense public overall healthiness factor and prostatic affliction may have a massive impression at the caliber of lifetime of many males. This publication will supply a handy precis for urologists, fundamental care physicians and nurse practitioners
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Additional resources for An illustrated pocketbook of prostatic diseases
Studies have revealed that, whereas many prostatic calculi are formed as a result of concretions of prostatic gland secretions, many are composed of similar constituents to those of renal, ureteric and bladder calculi (urinary constituents), thereby confirming the tendency of urine to enter the prostate. AN ILLUSTRATED POCKETBOOK OF PROSTATIC DISEASES 33 Figure 28 Prostatic calculi can be seen on a plain X-ray, perhaps secondary to intrapro-static urinary reflux. Calculi tend to form within the prostate, where they may constitute a nidus for chronic prostatic infection ' Figure 29 Prostatitis is characterized by an inflammatory response within the gland.
2 ml/s/year32 . Recent data confirm that men with larger prostates and higher PSA values suffer a faster rate of disease progression than those with smaller glands33. The explanation for these findings lies in the progressive expansion of the transitional zone by the adenoma (Figure 49). This process reduces the distensibility of the urethra during voiding and produces gradually increasing bladder outlet obstruction. This increase in prostate volume is associated with a progressive risk of lower urinary tract symptoms, and a negative impact on quality of life.
Transitional zone enlargement with concomitant increase in overall gland volume), lower urinary tract symptoms, and bladder outflow obstruction Figure 46 Although benign prostatic hyperplasia is the most common cause of lower urinary tract symptoms (LUTS), many other factors play a role AN ILLUSTRATED POCKETBOOK OF PROSTATIC DISEASES 47 Although bladder wall hypertrophy, which develops in response to the increased effort required during voiding, is associated with an increase in size and strength of detrusor smooth muscle bundles, there is also infiltration by collagen (Figure 47) and a relative depletion of parasympathetic nerve endings.